Job Description

Structural and functional alterations at the neuromuscular junction (NMJ) are observed in sarcopenia. NMJ changes have been shown to precede muscle atrophy and weakness, suggesting its role as an early event in sarcopenia pathogenesis. To better understand the underlying mechanisms of NMJ dysfunction, we seek innovative approaches using in vitro/ex vivo models to support mechanistic studies in sarcopenia. Potential in-scope solutions include establishing human and murine 2D and 3D models and mimicking aging using pharmacological, genetic or other approaches.

How would you propose modeling neuromuscular junction degeneration in sarcopenia to identify therapeutic targets or biomarkers? Get started on opnMe for a chance to join Boehringer Ingelheim as a fully funded PostDoc: https://lnkd.in/dNN9fEiK

Study the neuromuscular junction in sarcopenia

How would you propose to elucidate cellular and molecular mechanisms driving neuromuscular junction dysfunction in primary and secondary sarcopenia using in vitro models?

Apply for a PostDoc position at Boehringer Ingelheim in Biberach / Riss in Germany.
Submit your research proposal for a chance to be selected to conduct your proposed plan as part of your PostDoc project at one of the leading global pharmaceutical companies.

Call for proposals: All incoming applications accompanied by a research plan will be evaluated by a scientific jury, and, upon selection, you as the winner will have the opportunity to pursue your research project as defined by yourself as part of your PostDoc studies at Boehringer Ingelheim. An attractive package including salary, expenses, and additional company benefits will apply.